Decreased ejection fraction after implantation

Hello everybody!

I have 46 years old and already served a month with my PM implant because a degenerative disease of AV node. Yesterday made me a gamagramma, but it turned out that there is:

1. Slight apical defect that improves in stress in relation to recent PM

2. Dilated left ventricle (VTD 139ml, VTS 71ml) with borderline systolic function, LVEF 49%

3. Important asynchronous contraction of VI with a bandwidth of 240º, standard deviation of 60º, with the last viable contractile segment located in the middle lower side segment

In an echocardiogram that they did a month before implantation, my LVEF was 72% and there was no LV dilation, I have read that a poor placement of the electrode in the RV can cause the LVEF to drop, the LV to dilate and asynchrony occurs

Has something similar happened to anyone?

1 Comments

LV remodelling after prolonged RV-apical pacing

by crustyg - 2021-08-13 07:56:53

There's a whole flame-war waiting for the 'prolonged RV-apical pacing 'causes' LV-remodelling' - but this post isn't intended to trigger it.  The key word is prolonged.

AFAIK, LV remodelling due to / because of non-physiological pacing takes a lot longer to appear than you describe.

Unfortunately, I think it's much more likely that the same disease process that's damaged your AV-node is showing up in your ventricular muscle.

You should talk to your heart docs about this, and perhaps get a genetic test done.

I wish you well.

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